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Table 4 SSEP in different myoclonic subgroups

From: Electrophysiological characteristics and anatomical differentiation of epileptic and non-epileptic myoclonus

  Juvenile myoclonic epilepsy (N = 19) Progressive myoclonic epilepsy (N = 10) Secondary (post-hypoxic) Lance–Adams syndrome (N = 2) Secondary (post-ischemic stroke myoclonus) (N = 2) Secondary (hepatic) (N = 6) Secondary (chronic kidney) (N = 6) Drug-induced myoclonus (N = 5) P
Age 31.9 ± 12.5 34.4 ± 11.6 61 ± 4.5 56 ± 3.4 56.7 ± 5.9 59.3 ± 7.4 28.9 ± 10.9 0.13
N20 amplitude 1.9 ± 0.7 2.1 ± 0.6 0.9 ± 0.5 2.8 ± 0.7 1.9 ± 0.6 1.9 ± 0.8 1.9 ± 0.9 0.63
N20 latency 23.3 ± .18 23.2 ± .12 23 ± 2.4 23.4 ± 1.6 23.2 ± 0.1 23.3 ± 0.1 23.3 ± 0.1 0.22
P24 amplitude 2.6 ± 1.1 20.9 ± 27.5 10.6 ± 1.2 8.5 ± 0.6 7.8 ± 2.2 6.7 ± 1.6 6.9 ± 1.6 0.04*
P24 latency 25.2 ± 0.2 24.9 ± 0.55 24.4 ± 2.3 24.8 ± 1.7 25.5 ± 0.8 24.8 ± 0.6 24.9 ± 0.2 0.06
N33 amplitude 3.7 ± 1.4 24.5 ± 30.8 11.4 ± 1.3 12.5 ± 0.4 9.5 ± 2.1 8.8 ± 1.6 8.2 ± 2.1 0.04*
N33 latency 34.03 ± 0.1 33.54 ± 0.18 33.3 ± 1.8 35.6 ± 1.9 34.1 ± 0.6 34.1 ± 0.6 34 ± 0.21 0.14
P24–N33 complex amplitude 6.2 ± 2.5 45.4 ± 58.3 22.1 ± 1.2 21 ± 0.8 17.7 ± 4 15.6 ± 2.9 15.1 ± 3.6 0.04*
N20 amplitude 1.9 ± 0.7 2.01 ± 0.7 1.9 ± 0.5 1 ± 0.3 1.8 ± 0.6 2.3 ± 0.9 1.8 ± 0.8 0.7
N20 latency 23.2 ± 0.14 23.1 ± 0.11 23.6 ± 3.2 23.4 ± 0.7 23.7 ± 0.2 23.3 ± 0.1 23.2 ± 0.1 0.29
P24 amplitude 2.9 ± 1.2 19.4 ± 22.5 7.2 ± 0.7 8.4 ± 0.5 7.4 ± 2.9 7.1 ± 2.6 8.1 ± 2.1 0.02*
P24 latency 25.1 ± 0.1 25.2 ± 0.9 24.9 ± 3.2 25.1 ± 2.9 24.9 ± 0.3 25 ± 0.2 24.7 ± 0.2 0.39
N33 amplitude 4.1 ± 1.2 23.5 ± 26.5 9.6 ± 0.9 10.6 ± 0.8 9.1 ± 2.8 8.5 ± 2.8 10.1 ± 2.7 0.02*
N33 latency 34.1 ± 0.3 33.6 ± 0.57 33.70 ± 3.5 33.8 ± 2.2 34.3 ± .41 33.7 ± 0.4 34 ± 0.7 0.07
P24–N33 complex amplitude 6.9 ± 2.3 42.9 ± 48.9 16.8 ± 2.4 19 ± 2.3 16.5 ± 5 15.6 ± 5.3 18.2 ± 4.2 0.02*
EGG spike/polyspike wave complex 12 (63.2%) 10 (100%) 2 (100%) 2 (100%) 4 (66.6%) 4 (66.6%) 2 (40%) 0.02*